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The substantial gap between preclinical evidence and clinical validation represents the primary limitation for evidence-based clinical use

An agent that suppresses NF-B phosphorylation upstream of cytokine production could theoretically interrupt the inflammatory cascade before it causes epithelial damage

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Some researchers have conceptually explored combining both strategies for potentially synergistic effects on intracellular NAD+ levels, though no published data exists for this specific combination

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First, side effects are clearly dose-dependent, increasing as the dose goes up